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Section 6 of 6 · Open sections

Required section · Section 6 of 6

Keep the conclusion proportional

This case supports increased red-cell destruction with an intravascular component and morphology and DAT findings that warrant immune follow-up. It does not establish autoimmune hemolytic anemia, a complement-mediated disorder, a membrane defect, an enzyme deficiency, paroxysmal nocturnal hemoglobinuria (PNH), or thrombotic microangiopathy. Qualified clinical and specialized laboratory review remain necessary.

For an unresolved possible PNH pattern, refer to the qualified hematopathology or reference-laboratory pathway for validated PNH flow-cytometry interpretation.

Method and policy details change the action: validated analyzer and reagent, local intervals, hemolysis-index thresholds, DAT algorithm, controls, and referral criteria belong in the local procedure. CLIA requires applicable performance specifications and written procedures for calibrators, controls, calibration, reportable range, and corrective action. Use the validated local procedure for release, comments, redraw decisions, and referral.

Pattern-to-follow-up routing
ClueBounded laboratory actionLimit
IgG DAT with hemolysis patternQualified immunohematology reviewDAT does not establish immune hemolysis
Bite cellsQualified enzyme-testing reviewDoes not diagnose enzyme deficiency
Sickle cellsHemoglobin testing reviewMorphology alone is insufficient
Unexplained hemolysis, negative DATLocal repeat or reference DAT pathwayRoutine DAT can miss coating
Possible PNH patternValidated specialized flow referralNeeds clinical correlation

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What is the appropriate laboratory action for an unresolved possible PNH pattern?

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