Thyroid, rheumatoid and organ-specific autoantibodies
15 min
- Choose the antibody tests that fit a suspected organ-specific disease
- Tell thyroid antibody results from thyroid function, and TRAb binding from stimulation
- Tell a nonspecific rheumatoid factor result from a more specific anti-CCP result
Try first
Get the idea
Pair the antibody with the organ
An organ-specific autoantibody is most useful beside evidence from the organ itself: hormone levels, enzymes, function tests or tissue findings.1 Each suspected disease has its own first antibody.
| Suspected disease | First antibody tests |
|---|---|
| Hashimoto thyroiditis | Anti-thyroid peroxidase (anti-TPO) |
| Graves disease | TSH receptor antibody (TRAb) |
| Celiac disease | IgA anti-tTG with total IgA |
| Autoimmune hepatitis type 1 | ANA and smooth muscle antibody |
| Primary biliary cholangitis | Antimitochondrial antibody |
| Rheumatoid arthritis | Rheumatoid factor and anti-CCP |
Celiac serology needs IgA
IgA anti-tTG is the first celiac test in a patient eating gluten. A patient with IgA deficiency cannot make IgA anti-tTG, so every IgA screen is paired with a total IgA. When total IgA is very low, an IgG-based test such as IgG anti-deamidated gliadin peptide follows. A gluten-free diet lowers the antibody and can make any celiac test falsely negative.4
Thyroid antibodies name a cause
Thyroid function comes from TSH and free T4. Anti-TPO supports Hashimoto thyroiditis, and TRAb supports Graves disease. A positive antibody names a likely cause and says nothing about whether the gland is overactive, underactive or working normally today.2
TRAb comes in two formats. A binding assay detects any antibody that attaches to the TSH receptor, whether it stimulates or blocks the receptor. A bioassay measures what the antibody does to receptor-bearing cells, and only a bioassay reports stimulating activity. The report names which format was used.2
Rheumatoid factor and anti-CCP
Rheumatoid factor is usually IgM against the Fc region of IgG. It appears in other inflammatory diseases, in infections and in some healthy people, especially older adults. Anti-cyclic citrullinated peptide (anti-CCP) antibody is more specific for rheumatoid arthritis and can be positive when RF is negative. Neither antibody alone establishes the disease or its current activity. Both are read with the joint findings.1,3
References
- Abbas AK, Lichtman AH, Pillai S, Henrickson S. Cellular and Molecular Immunology. 11th ed. Elsevier; 2025. Elsevier.
- ARUP Laboratories. Autoimmune thyroiditis. ARUP Consult. Accessed September 27, 2026.
- ARUP Laboratories. Rheumatoid arthritis. ARUP Consult. Accessed September 27, 2026.
- Rubio-Tapia A, Hill ID, Kelly CP, Calderwood AH, Murray JA. American College of Gastroenterology guidelines update: diagnosis and management of celiac disease. Am J Gastroenterol. 2023;118(1):59-76. doi:10.14309/ajg.0000000000002075
- ARUP Laboratories. Primary biliary cholangitis. ARUP Consult. Accessed September 27, 2026.
Watch one
Oskar Lindqvist, 27, is a line cook who tastes everything he makes, bread included. His provider orders a celiac screen for chronic diarrhea and iron deficiency.
Can the negative IgA anti-tTG be reported as a negative celiac screen?
| Test | Result | Previous | Reference interval | Flag |
|---|---|---|---|---|
| IgA anti-tTG | 0.8 U/mL | <4.0 U/mL | ||
| Total IgA | <5 mg/dL | 70–400 mg/dL | Low | |
| IgG | 1,080 mg/dL | 700–1,600 mg/dL | ||
| IgM | 115 mg/dL | 40–230 mg/dL |
Specimen: H 3, L 12, I 1. Serum, collected 08:40
- Check the history first. He eats gluten every day, so the screen was done at a useful time.
Celiac antibodies fall when gluten leaves the diet, so every screen is read against intake.
- Read the total IgA next. It is below 5 mg/dL, far under the reference interval.
The IgA screen depends on the patient's ability to make IgA.
- Check the other immunoglobulins. IgG and IgM are both within their reference intervals.
IgG and IgM within their intervals point to a selective loss of IgA.
- Now read the anti-tTG. The 0.8 U/mL result is uninformative in this patient.
A patient who makes almost no IgA cannot make IgA anti-tTG.
- Choose the follow-up. An IgG-based test, such as IgG anti-deamidated gliadin peptide, follows under the laboratory's algorithm.
An IgG-based antibody does not depend on IgA production.
Your turn
Use it
- Delphine Okafor, 46, is a pastry chef who has stopped kneading dough by hand because her fingers ache and swell every morning.
- She has also lost weight and feels her heart racing.
- Her provider orders joint and thyroid serology.
- The laboratory's TRAb method is a receptor-binding immunoassay.
| Test | Result | Previous | Reference interval | Flag |
|---|---|---|---|---|
| Rheumatoid factor | 9 IU/mL | <14 IU/mL | ||
| Anti-CCP | 86 U/mL | <20 U/mL | High | |
| TSH | 0.02 mIU/L | 0.40–4.50 mIU/L | Low | |
| Free T4 | 3.1 ng/dL | 0.8–1.8 ng/dL | High | |
| TSH receptor antibody | 6.4 IU/L | ≤1.75 IU/L | High |
Specimen: H 2, L 9, I 1. Serum, collected 09:15
The clue that settled this case is the method behind each antibody. Anti-CCP is the specific rheumatoid marker, so a negative RF does not undo it. The TRAb came from a binding assay, so the report states binding and leaves stimulating activity unmeasured. TSH and free T4 describe what her thyroid is doing.
Results
- Choose the antibody tests that fit a suspected organ-specific disease
- Tell thyroid antibody results from thyroid function, and TRAb binding from stimulation
- Tell a nonspecific rheumatoid factor result from a more specific anti-CCP result
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